![]() If you have retinitis pigmentosa there are some devices that can help make objects look brighter and larger, such as low-vision magnifiers. Research has shown some promise with a combination of vitamin A, lutein and oily fish high in the omega-3 fatty acid DHA. While there is currently no cure for retinitis pigmentosa, researchers are looking into a range of treatment options, such as stem cells, medications, gene therapy and transplants. Approximately 100,000 Americans have retinitis pigmentosa. Retinitis pigmentosa diagnosis occurs anywhere from early infancy to late adulthood. Sometimes the disorder is classified by the age of onset or the inheritance pattern. There are many forms of retinitis pigmentosa. In the late stages of the disease, there is only a small area of central vision remaining. The ability to see color is eventually lost. As the condition worsens, peripheral vision disappears, which results in tunnel vision. Because the rods are primarily affected, it becomes harder to see in dim light, thus causing a loss of night vision. In retinitis pigmentosa, the photoreceptors (primarily the rods) begin to deteriorate and lose their ability to function. Another layer of the retina is called the retinal pigmented epithelium (RPE). The rod and cone photoreceptors convert light into electrical impulses and send the message to the brain via the optic nerve. The number of rods increases in the periphery. The area surrounding the fovea contains the rods, which are necessary for peripheral vision and night vision. The cones are responsible for sharp, central vision and color vision and are primarily located in a small area of the retina called the fovea. ![]() One layer contains two types of photoreceptor cells referred to as the rods and cones. The retina lines the interior surface of the back of the eye. Retinitis pigmentosa is a chronic, hereditary eye disease characterized by black pigmentation and gradual degeneration of the retina.
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